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Figures
Fig. 1
Normal sinus rhythm and a heart rate of 68 beats/min; electrocardiogram on admission.
Fig. 2
Diffuse intraparenchymal, subdural as plastering style, and intraventricular (in lateral and third ventricles) hemorrhage in the left hemisphere; CT obtained on admission.
Fig. 3
Diffuse intraparenchymal and intraventricular hemorrhage foci, and pneumocephalus and craniotomy defect depending on the operation; CT obtained postoperative first day (an increase in the amount of hemorrhage).
Fig. 4
Diffuse intraparenchymal and intraventricular hemorrhage areas and craniotomy defect depending on the operation; CT obtained postoperative fourth day (an increase in the amount of hemorrhage).
Fig. 5
Diffuse intraparenchymal and intraventricular hemorrhage areas and craniotomy defect depending on the operation and vasogenic edema areas around the hemorrhage; CT obtained postoperative seventh day (a decrease in the amount of intraventricular hemorrhage and an increase in the amount of intraparenchymal and vasogenic edema).
Fig. 6
Intraparenchymal and intraventricular hemorrhage, vasogenic edema, and craniotomy defect; CT obtained postoperative 12th day (a decrease in the amount of intraparenchymal and intraventricular hemorrhage and an increase in the amount of vasogenic edema).
Bleeding, the most frightening adverse effect of anticoagulants, may occur in different parts of the body. When intracerebral hemorrhage in individuals used anticoagulant drugs is compared with normal coagulation function, the volume of bleeding is increased and the prognosis is worse. There are few studies in the literature regarding the presence of intracerebral hemorrhage and the volume and prognosis of bleeding associated with rivaroxaban, a new oral anticoagulant. Therefore, the clinical and radiologic findings and follow-up of an 80-year-old male patient with intracerebral hemorrhage who uses rivaroxaban for anticoagulation are presented in this article.
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