Dabigatran-related coagulopathy: when can we assume the effect has “worn off”?
Charles V. Pollack Jr.
,x
, MA, MDCharles V. Pollack
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Affiliations
- Department of Emergency Medicine, Pennsylvania Hospital, Philadelphia, PA, USA
Correspondence
- Corresponding author.

x
Charles V. Pollack
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Affiliations
- Department of Emergency Medicine, Pennsylvania Hospital, Philadelphia, PA, USA
Correspondence
- Corresponding author.
Jerrold H. Levy
,x
, MDJerrold H. Levy
John Eikelboom
,x
, MDJohn Eikelboom
Jeffrey I. Weitz
,x
, MDJeffrey I. Weitz
Frank W. Sellke
,x
, MDFrank W. Sellke
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Affiliations
- Brown University Medical School and Lifespan Hospitals, Providence, RI, USA
Menno V. Huisman
,x
, MD, PhDMenno V. Huisman
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Affiliations
- Leiden University Medical Center, Leiden, The Netherlands
Thorsten Steiner
,x
, MD, PhDThorsten Steiner
Pieter Kamphuisen
,x
, MD, PhDPieter Kamphuisen
Richard A. Bernstein
x
, MD, PhDRichard A. Bernstein
Article Info
Publication History
Published Online: August 28, 2014Accepted: August 25, 2014; Received: August 24, 2014;
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This issue of American Journal of Emergency Medicine presents a timely report that should be noted by all practicing emergency physicians. Nzwalo et al [1] describe a 61-year-old man with nonvalvular atrial fibrillation, taking 150 mg dabigatran twice daily for stroke protection, who presented to their emergency department (ED) with an acute ischemic stroke (AIS). The patient had a normal creatinine clearance and a normal thrombin time (TT). He was treated with intravenous recombinant tissue plasminogen activator (rt-PA) with good neurologic outcome and no bleeding complications.
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© 2014 Elsevier Inc. Published by Elsevier Inc. All rights reserved.
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